A <a href="https://www.mid-day.com/news/world-news/article/iran-enforces-new-sovereign-transit-rules-in-strait-of-hormuz-mandates-prior-permits-for-vessels-23629124" rel="nofollow">new drug</a> kept mice from becoming obese even after long-term exposure to a sugar-rich, fat-heavy diet.
The work points to magnesium movement inside mitochondria, the energy-producing structures found in cells.&nbsp;
When magnesium enters mitochondria through a gene called MRS2, it can slow the cell&rsquo;s ability to burn sugar and fat. Researchers removed the MRS2 gene in mice.&nbsp;
After 52 weeks of a fat-heavy diet, normal mice gained weight and developed signs of metabolic disease, while mice without MRS2 stayed lean.
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Researchers have developed a new drug approach that prevented mice from becoming obese even after prolonged exposure to a high-fat, sugar-rich diet

Researchers find potential obesity treatment that helped mice stay lean despite long-term high-fat, high-sugar diet

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Researchers have developed a new drug approach that prevented mice from becoming obese even after prolonged exposure to a high-fat, sugar-rich diet. The study focused on blocking the MRS2 gene linked to magnesium movement inside mitochondria, helping cells burn fat and sugar more efficiently

New drug prevents obesity even on high-fat diet

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